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A non-steroidal anti-inflammatory drug or NSAID is used to relieve some symptoms caused by arthritis, such as inflammation, swelling, stiffness, and joint pain. In addition to their pain killing effect they also, as the name suggests, have an anti-inflammatory action – this is what makes them so useful in arthritis where much of the pain is generated by inflammation in the joint.

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The reason NSAIDs have several beneficial effects is due to a class of compounds produced by the body called prostaglandins. These are are mainly involved in providing protection for various body tissues. Different prostaglandins do this in different ways but a particular group encourages the inflammatory response and sensitizes nerve endings to enhance pain sensation. Believe it or not this is a protective action: inflammation although unpleasant is important in protecting against infection and pain of course alerts us to an injury.

NSAIDs interfere with the production of prostaglandins by inhibiting enzymes called cyclooxygenases. Two are of relevance here and are abbreviated to COX1 and COX2. By limiting prostaglandin production NSAIDs limit inflammation and pain sensation and keep fever down. Most NSAIDs inhibit the clotting of blood for only a few hours. Although NSAIDs have a similar mechanism of action, individuals who do not respond to one NSAID may respond to another.